Idiopathic Pulmonary Fibrosis (IPF) Therapeutics
Antineoplastics
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Idiopathic Pulmonary Fibrosis (IPF) Therapeutics
Idiopathic pulmonary fibrosis (IPF) therapeutics include APIs developed to slow disease progression, reduce fibrotic tissue formation, and improve respiratory function in patients with chronic and progressive lung fibrosis. Key agents include pirfenidone and nintedanib, which inhibit fibrogenic cytokines, growth factor signaling, and pathways involving TGF-β, VEGF, and PDGF. These drugs reduce fibroblast proliferation, collagen deposition, and inflammatory signaling in lung tissue. Although IPF remains incurable, these therapies significantly delay functional decline and increase quality of life. IPF therapeutics are essential for managing symptoms, reducing exacerbations, and extending survival in affected patients. Ongoing research focuses on combination regimens, antifibrotic biologics, and precision-medicine approaches.
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CAS No.:100992-59-4
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CAS No.:1254472-97-3
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CAS No.:1448428-04-3
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CAS No.:1448428-05-4
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CAS No.:1450824-22-2
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CAS No.:1488354-15-9
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